英作文の添削をお願いしたいです。文法的な問題だけではなくロジックとして
英作文の添削をお願いしたいです。文法的な問題だけではなくロジックとしておかしいところがあればご指摘ください。長文ですがよろしくお願いいたします。
The number of patient with diabetes has continued to rise recently in Japan. In 2006, the incidence of diabetes increased by 800 thousand in 2002 to 8.2 million, and patients with impaired glucose tolerance bring the number to 18.7 million. Also, it is problem that there are a lot of patients with complications such as nephropathy and retinopathy. Additionally, since the incidence of cerebral infarction increased 2-4 times, that of arteriosclerosis has been seen as a problem.
Insulin which is secreted from pancreatic β cell stimulates uptake of blood glucose into the cell. In healthy people, postprandial glucose level is achieved a peak after 30 minutes, and returned to prior level after 120 minutes. But, as the presence of either inadequate insulin secretion or insulin resistance suppresses glucose uptake or take time than usual, hyperglycemic state is continued and excess glucose in blood is excreted into urine. Insulin abnormality is caused by various factors ranged from genetic deficiency to pregnancy, and the main two types of diabetes are type 1 diabetes with absolute insulin deficiency and type 2 diabetes with relative insulin deficiency or abnormality of secretion. In Japan, most of diabetic patients are categorized into type 2 diabetes which is caused by environment factor such as obese, immobilization, stress and overeating. Adipocytes secrete various adipocytekines including both good and bad for insulin sensitivity. Among these, only adiponectin is good for insulin sensitivity. But secretion of adiponectin is suppressed and TNF-αwhich inhibits insulin signal as well as expression of receptors associated with insulin signal and GLUT4 is more secreted by enlarged adipocytes in obese individual, especially with more visceral fat. つづく・・・
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